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Ischemic proximal tubular injury primes mice to endotoxin-induced TNF-α generation and systemic release

Author
ZAGER, R. A1 ; JOHNSON, Ali C. M1 ; HANSON, Sherry Y1 ; LUND, Steve1
[1] Department of Medicine, University of Washington, and the Fred Hutchinson Cancer Research Center, Seattle, Washington, United States
Source

American journal of physiology. Renal physiology. 2005, Vol 58, Num 2, pp F289-F297 ; ref : 34 ref

ISSN
1931-857X
Scientific domain
Physiology, morphology; Urology, nephrology
Publisher
American Physiological Society, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Author keyword
HK-2 cells antimycin A proximal tubules sepsis syndrome tumor necrosis factor
Keyword (fr)
Appareil urinaire Endotoxine Facteur nécrose tumorale α Facteur nécrose tumorale Ischémie Libération Lésion Septique syndrome Souris Tube proximal Appareil circulatoire pathologie Cytokine Infection Mammalia Rein Rodentia Toxine Vertebrata
Keyword (en)
Urinary system Endotoxin Tumor necrosis factor α Tumor necrosis factor Ischemia Release Lesion Sepsis syndrome Mouse Proximal tube Cardiovascular disease Cytokine Infection Mammalia Kidney Rodentia Toxin Vertebrata
Keyword (es)
Aparato urinario Endotoxina Factor necrosis tumoral α Factor necrosis tumoral Isquemia Liberación Lesión Séptico síndrome Ratón Tubo proximal Aparato circulatorio patología Citoquina Infección Mammalia Riñón Rodentia Toxina Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A18 Vertebrates: urinary system

Discipline
Vertebrates : digestive and urinary system
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
16968762

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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