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Differential induction of apoptosis in B16 melanoma and EL-4 lymphoma cells by cytostatin and bactobolin

Author
KAWADA, M1 ; AMEMIYA, M1 ; ISHIZUKA, M1 ; TAKEUCHI, T1
[1] Institute for Chemotherapy, M. C. R. F., 18-24 Aza-Motono, Miyamoto, Numazu, Shizuoka 410-0301, Japan
Source

Japanese journal of cancer research. 1999, Vol 90, Num 2, pp 219-225 ; ref : 24 ref

CODEN
GANNA2
ISSN
0910-5050
Scientific domain
Medical oncology
Publisher
Japanese Cancer Association, Tokyo
Publication country
Japan
Document type
Article
Language
English
Keyword (fr)
Animal Anticancéreux Cycline In vitro Interleukin 1-β converting enzyme Lignée cellulaire établie Lymphome EL4 Mécanisme action Mélanome B16 Souris Spontané Cysteine endopeptidases Enzyme Hydrolases Hémopathie maligne Lymphoprolifératif syndrome Mammalia Peptidases Rodentia Tumeur maligne Vertebrata
Keyword (en)
Animal Antineoplastic agent Cyclin In vitro Interleukin 1-β converting enzyme Established cell line EL4-Lymphoma Mechanism of action B16-Melanoma Mouse Spontaneous Cysteine endopeptidases Enzyme Hydrolases Malignant hemopathy Lymphoproliferative syndrome Mammalia Peptidases Rodentia Malignant tumor Vertebrata
Keyword (es)
Animal Anticanceroso Cyclina In vitro Interleukin 1-β converting enzyme Línea celular establecida Linfoma EL4 Mecanismo acción Melanoma B16 Ratón Espontáneo Cysteine endopeptidases Enzima Hydrolases Hemopatía maligna Linfoproliferativo síndrome Mammalia Peptidases Rodentia Tumor maligno Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02R Antineoplastic agents / 002B02R01 General aspects

Discipline
Pharmacological treatments
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1702175

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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