Nuclear translocation of UDCA by the glucocorticoid receptor is required to reduce TGF-β1-induced apoptosis in rat hepatocytes
- Author
- SOLA, Susana1 2 ; AMARAL, Joana D1 ; CASTRO, Rui E1 ; RAMALHO, Rita M1 ; BORRALHO, Pedro M1 ; KREN, Betsy T2 ; TANAKA, Hirotoshi3 ; STEER, Cifford J2 4 ; RODRIGUES, Cecilia M. P1
[1] Centro de Patogénese Molecular, Faculty of Pharmacy, University of Lisbon, Lisbon, Portugal
[2] Department of Medicine, University of Minnesota Medical School, Minneapolis, MN, United States
[3] Division of Clinical Immunology, Advanced Clinical Research Center, Institute of Medical Science, University of Tokyo, Tokyo, Japan
[4] Department of Genetics, Cell Biology, and Development, University of Minnesota Medical School, Minneapolis, MN, United States - Source
Hepatology (Baltimore, Md.). 2005, Vol 42, Num 4, pp 925-934, 10 p ; ref : 45 ref
- CODEN
- HPTLD9
- ISSN
- 0270-9139
- Scientific domain
- Gastroenterology
- Publisher
- Wiley, Hoboken, NJ
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Acide biliaire Acide ursodéoxycholique Animal Apoptose Cytosol Facteur croissance transformant β1 Gastroentérologie Hépatocyte Microscopie confocale Rat Récepteur glucocorticoïde Translocation Mammalia Rodentia Vertebrata
- Keyword (en)
- Bile acid Ursodeoxycholic acid Animal Apoptosis Cytosol Transforming growth factor β1 Gastroenterology Hepatocyte Confocal microscopy Rat Glucocorticoid receptor Translocation Mammalia Rodentia Vertebrata
- Keyword (es)
- Acido biliar Acido ursodeoxicólico Animal Apoptosis Citosol Factor crecimiento transformante β1 Gastroenterología Hepatocito Microscopía confocal Rata Receptor glucocorticoide Translocación Mammalia Rodentia Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B13 Gastroenterology. Liver. Pancreas. Abdomen
- Discipline
- Gastroenterology. Liver. Pancreas. Abdomen
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 17165461
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1002/hep.20870 
https://dx.doi.org/10.1002/hep.20870
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