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Intravenous IGF-I receptor antisense reduces IGF-IR expression and diminishes pressor responses to angiotensin II in conscious normotensive rats

Author
TIEN THUY NGUYEN1 ; WHITE, Paul James1
[1] Department of Pharmaceutical Biology and Pharmacology, Victorian College of Pharmacy, Monash University, Parkville, Victoria 3052, Australia
Source

British journal of pharmacology. 2005, Vol 146, Num 7, pp 935-941, 7 p ; ref : 38 ref

CODEN
BJPCBM
ISSN
0007-1188
Scientific domain
Pharmacology drugs
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
Cardiovascular angiotensin II antisense insulin-like growth factor-I receptor insulin-like growth factor-I
Keyword (fr)
Angiotensine II Animal Appareil circulatoire Facteur croissance IGF1 Facteur croissance IGF Rat Récepteur biologique Voie intraveineuse Hormone peptide Mammalia Octapeptide Rodentia Système renin angiotensine Vertebrata
Keyword (en)
Angiotensin II Animal Circulatory system Insulin like growth factor 1 Insulin like growth factor Rat Biological receptor Intravenous administration Peptide hormone Mammalia Octapeptide Rodentia Renin angiotensin system Vertebrata
Keyword (es)
Angiotensina II Animal Aparato circulatorio Factor crecimiento IGF1 Factor crecimiento IGF Rata Receptor biológico Vía intravenosa Hormona péptido Mammalia Octapéptido Rodentia Sistema renin angiotensina Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments

Discipline
General pharmacology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
17299641

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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