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The involvement of the apoptosis-modulating proteins ERK 1/2, Bcl-xL and Bax in the resolution of acute inflammation in vivo

Author
SAWATZKY, Deborah A1 2 ; WILLOUGHBY, Derek A1 ; COLVILLE-NASH, Paul R1 ; ROSSI, Adriano G1 2
[1] Department of Experimental Pathology, St. Bartholomew's, and the Royal London Medical School, London, United Kingdom
[2] Medical Research Council Centre for Inflammation Research, The Queen's Medical Research Institute, University of Edinburgh Medical School, Edinburgh, United Kingdom
Source

The American journal of pathology. 2006, Vol 168, Num 1, pp 33-41, 9 p ; ref : 60 ref

CODEN
AJPAA4
ISSN
0002-9440
Scientific domain
Medical oncology; Medical laboratory techniques; General medicine general surgery
Publisher
American Society for Investigative Pathology, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Aigu Anatomopathologie Apoptose Extracellular signal-regulated protein kinase In vivo Inflammation Mitogen-activated protein kinase Mort cellulaire Protéine Bcl2 Enzyme
Keyword (en)
Acute Anatomic pathology Apoptosis Extracellular signal-regulated protein kinase In vivo Inflammation Mitogen-activated protein kinase Cell death Enzyme
Keyword (es)
Agudo Anatomía patológica Apoptosis Extracellular signal-regulated protein kinase In vivo Inflamación Mitogen-activated protein kinase Muerte celular Enzima
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B24 Investigative techniques, diagnostic techniques (general aspects) / 002B24O Pathology. Cytology. Biochemistry. Spectrometry. Miscellaneous investigative techniques

Discipline
Scanning and diagnostic techniques
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
17402015

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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