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Severe liver degeneration in mice lacking the IκB kinase 2 gene

Author
QIUTANG LI1 ; VAN ANTWERP, D1 ; MERCURIO, F2 ; LEE, K.-F1 ; VERMA, I. M1
[1] Salk Institute, La Jolla, CA 92037, United States
[2] Signal Pharmaceuticals, San Diego, CA 92121, United States
Source

Science (Washington, D.C.). 1999, Vol 284, Num 5412, pp 321-325 ; ref : 20 ref

CODEN
SCIEAS
ISSN
0036-8075
Scientific domain
Multidisciplinary
Publisher
American Association for the Advancement of Science, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Apoptose Chaîne peptidique β Ciblage Développement embryonnaire Facteur nécrose tumorale Foie Inflammation Interleukine 1 Mutation Phosphorylation Souris Transduction signal Facteur transcription NFκB Gène Ikk2 IκB kinase Protéine IKK Protéine IκB Mammalia Rodentia Vertebrata
Keyword (en)
Apoptosis Beta-Peptide chain Targeting Embryonic development Tumor necrosis factor Liver Inflammation Interleukin 1 Mutation Phosphorylation Mouse Signal transduction Transcription factor NFκB Mammalia Rodentia Vertebrata
Keyword (es)
Apoptosis Cadena peptídica β Blancado Desarrollo embrionario Factor necrosis tumoral Hígado Inflamación Interleuquina 1 Mutación Fosforilación Ratón Transducción señal Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A07 Genetics of eukaryotes. Biological and molecular evolution / 002A07C Classical genetics, quantitative genetics, hybrids / 002A07C01 Animals / 002A07C01B Vertebrata

Discipline
Eukaryotes genetics. Biological and molecular evolution
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1746247

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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