Modulation of STAT1 protein levels : a mechanism shaping CD8 T-cell responses invivo
- Author
- DURBIN, E (Commentator (written text))
1 ; PILAR GIL, M2 ; SALOMON, Rachelle2 ; LOUTEN, Jennifer2 ; BIRON, Christine A2
[1] OHIO STATE UNIVERSITY, United States
[2] Department of Molecular Microbiology and Immunology, Division of Biology and Medicine, Brown University, Providence, RI, United States - Source
Blood. 2006, Vol 107, Num 3, pp 847-848, 9 p ; ref : 41 ref
- ISSN
- 0006-4971
- Scientific domain
- Medical oncology; Hematology
- Publisher
- The Americain Society of Hematology, Washington, DC
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Animal Antiviral Facteur transcription STAT1 Immunorégulation Interféron alpha Interféron bêta Lymphocyte T Mécanisme action Réponse immune Souris Antigène CD8 Voie de signalisation Cytokine Mammalia Rodentia Vertebrata
- Keyword (en)
- Animal Antiviral Transcription factor STAT1 Immune regulation Alpha interferon Beta interferon T-Lymphocyte Mechanism of action Immune response Mouse Signaling pathway Cytokine Mammalia Rodentia Vertebrata
- Keyword (es)
- Animal Antiviral Factor transcripción STAT1 Inmunoregulación Interferón alfa Interferón beta Linfocito T Mecanismo acción Respuesta inmune Ratón Citoquina Mammalia Rodentia Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C08 Modulation of the immune response (stimulation, suppression)
- Discipline
- Fundamental immunology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 17489679
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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