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Hypoxia stimulates breast carcinoma cell invasion through MT1-MMP and MMP-2 activation

Author
MUNOZ-NAJAR, U. M1 ; NEURATH, K. M1 ; VUMBACA, F1 ; CLAFFEY, K. P1
[1] Center for Vascular Biology, University of Connecticut Health Center, Farmington, CT, United States
Source

Oncogene (Basingstoke). 2006, Vol 25, Num 16, pp 2379-2392, 14 p ; ref : 1 p.1/4

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
MT1- MMP RhoA breast cancer hypoxia invasion
Keyword (fr)
Activation Carcinogenèse Carcinome sein Gelatinase A Hypoxie Interstitial collagenase Oxygène dGTPase Cancer sein Enzyme Esterases Glande mammaire pathologie Hydrolases Metalloendopeptidases Peptidases Triphosphoric monoester hydrolases Tumeur maligne
Keyword (en)
Activation Carcinogenesis Breast carcinoma Gelatinase A Hypoxia Interstitial collagenase Oxygen dGTPase Breast cancer Enzyme Esterases Mammary gland diseases Hydrolases Metalloendopeptidases Peptidases Triphosphoric monoester hydrolases Malignant tumor
Keyword (es)
Activación Carcinogénesis Carcinoma pecho Gelatinase A Hipoxia Interstitial collagenase Oxígeno dGTPase Cáncer pecho Enzima Esterases Glándula mamaria patología Hydrolases Metalloendopeptidases Peptidases Triphosphoric monoester hydrolases Tumor maligno
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B20 Gynecology. Andrology. Obstetrics / 002B20E Mammary gland diseases / 002B20E02 Tumors

Discipline
Gynecology. Andrology. Obstetrics Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
17701574

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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