Insulin-like growth factor-1 and PTEN deletion enhance cardiac L-type Ca2+ currents via increased PI3Kα/PKB signaling
- Author
- HUI SUN1 ; KERFANT, Benoit-Gilles1 ; DONGLING ZHAO1 ; TRIVIERI, Maria G1 2 ; OUDIT, Gavin Y1 2 ; PENNINGER, Josef M3 ; BACKX, Peter H2 4
[1] Departments of Physiology and Medicine, University Health Network, Canada
[2] Division of Cardiology, University Health Network, Canada
[3] Institute for Molecular Biotechnology, Austrian Academy of Science, Vienna, Austria
[4] Heart & Stroke/Richard Lewar Centre of Excellence in Cardiovascular Research, University of Toronto, Canada - Source
Circulation research. 2006, Vol 98, Num 11, pp 1390-1397, 8 p ; ref : 43 ref
- CODEN
- CIRUAL
- ISSN
- 0009-7330
- Scientific domain
- Cardiology, blood circulation, phlebology
- Publisher
- Lippincott, Hagerstown, MD
- Publication country
- United States
- Document type
- Article
- Language
- English
- Author keyword
- Akt/PKB L-type Ca2+ channels PI3K PTEN isoforms
- Keyword (fr)
- Appareil circulatoire Calcium Délétion Facteur croissance IGF1 Forme moléculaire Mammalia Transduction signal Canal calcique L Elément minéral Vertebrata
- Keyword (en)
- Circulatory system Calcium Deletion Insulin like growth factor 1 Molecular form Mammalia Signal transduction L-type calcium channel Inorganic element Vertebrata
- Keyword (es)
- Aparato circulatorio Calcio Deleción Factor crecimiento IGF1 Forma molecular Mammalia Transducción señal Canal calcio tipo L Elemento inorgánico Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A22 Vertebrates: cardiovascular system
- Discipline
- Vertebrates : cardiovascular system
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 17860236
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1161/01.RES.0000223321.34482.8c 
https://dx.doi.org/10.1161/01.RES.0000223321.34482.8c
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