Silencing of epithelial CXCL12 expression by DNA hypermethylation promotes colonic carcinoma metastasis
- Author
- WENDT, M. K1 ; JOHANESEN, P. A1 ; KANG-DECKER, N2 ; BINION, D. G3 ; SHAH, V2 ; DWINELL, M. B1
[1] Department of Microbiology and Molecular Genetics, Medical College of Wisconsin, Milwaukee, WI, United States
[2] GI Research Unit, Department of Medicine, Mayo Clinic Foundation, Rochester, MN, United States
[3] Department of Medicine, Medical College of Wisconsin, Milwaukee, WI, United States - Source
Oncogene (Basingstoke). 2006, Vol 25, Num 36, pp 4986-4997, 12 p ; ref : 46 ref
- ISSN
- 0950-9232
- Scientific domain
- Cell biology, histology; Medical oncology; Genetics
- Publisher
- Nature Publishing, Basingstoke
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Author keyword
- chemokine epigenetic epithelial cell intestinal mucosa
- Keyword (fr)
- Carcinogenèse Carcinome Cellule épithéliale Chimiokine DNA Epigénétique Epithélium Expression génique Muqueuse Métastase Méthylation Tumeur maligne
- Keyword (en)
- Carcinogenesis Carcinoma Epithelial cell Chemokine DNA Epigenetics Epithelium Gene expression Mucosa Metastasis Methylation Malignant tumor
- Keyword (es)
- Carcinogénesis Carcinoma Célula epitelial Quimioquina DNA Epigenética Epitelio Expresión genética Mucosa Metástasis Metilación Tumor maligno
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes
- Discipline
- Molecular and cell biology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 18031523
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1038/sj.onc.1209505 
https://dx.doi.org/10.1038/sj.onc.1209505
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