Clinical implications of the mechanism of epidermal growth factor receptor inhibitors
- Author
- MARSHALL, John1
[1] Division of Hematology/Oncology, Lombardi Cancer Center, Georgetown University Medical Center, Washington, DC, United States - Source
Cancer. 2006, Vol 107, Num 6, pp 1207-1218, 12 p ; ref : 114 ref
- CODEN
- CANCAR
- ISSN
- 0008-543X
- Scientific domain
- Medical oncology
- Publisher
- Wiley-Liss, New York, NY
- Publication country
- United States
- Document type
- Article
- Language
- English
- Author keyword
- cetuximab drug toxicity drug-dose response relationship epidermal growth factor receptor erlotinib
- Keyword (fr)
- Anticancéreux Cancérologie Cétuximab Erlotinib Mécanisme Médicament Relation dose réponse Récepteur facteur croissance épiderme Récepteur facteur croissance Toxicité Inhibiteur de la tyrosine kinase Anticorps monoclonal Enzyme Inhibiteur enzyme Protein-tyrosine kinase Quinazoline dérivé Transferases
- Keyword (en)
- Antineoplastic agent Cancerology Cetuximab Erlotinib Mechanism Drug Dose activity relation Epidermal growth factor receptor Growth factor receptor Toxicity Monoclonal antibody Enzyme Enzyme inhibitor Protein-tyrosine kinase Quinazoline derivatives Transferases
- Keyword (es)
- Anticanceroso Cancerología Cetuximab Erlotinib Mecanismo Medicamento Relación dosis respuesta Receptor factor crecimiento epidermi Receptor factor crecimiento Toxicidad Anticuerpo monoclonal Enzima Inhibidor enzima Protein-tyrosine kinase Transferases
- Classification
- Discipline
- Tumours
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 18099386
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1002/cncr.22133 
https://dx.doi.org/10.1002/cncr.22133
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