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Effect of AT2 receptor blockade on the pathogenesis of renal fibrosis

Author
MORRISSEY, J. J1 ; KLAHR, S1
[1] Renal Division of the Department of Internal Medicine, and the Department of Cell Biology-Physiology, Washington University School of Medicine and Barnes-Jewish Hospital, St. Louis, Missouri 63110, United States
Source

American journal of physiology. Renal physiology. 1999, Vol 45, Num 1, pp F39-F45 ; ref : 36 ref

ISSN
1931-857X
Scientific domain
Physiology, morphology; Urology, nephrology
Publisher
American Physiological Society, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Actine Angiotensine II Apoptose Expression génique Macrophage Monocyte Muscle lisse Mécanisme action Rat Rein pathologie Récepteur peptidergique Appareil urinaire pathologie Mammalia Rodentia Système renin angiotensine Vertebrata
Keyword (en)
Actin Angiotensin II Apoptosis Gene expression Macrophage Monocyte Smooth muscle Mechanism of action Rat Kidney disease Peptidergic receptor Urinary system disease Mammalia Rodentia Renin angiotensin system Vertebrata
Keyword (es)
Actina Angiotensina II Apoptosis Expresión genética Macrófago Monocito Músculo liso Mecanismo acción Rata Riñón patología Receptor peptidérgico Aparato urinario patología Mammalia Rodentia Sistema renin angiotensina Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1815725

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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