Estradiol increases apoptosis in human coronary artery endothelial cells by up-regulating fas and fas ligand expression
- Author
- SELI, Emre1 ; GUZELOGLU-KAYISLI, Ozlem1 ; CAKMAK, Hakan1 ; KAYISLI, Umit A1 ; SELAM, Belgin1 ; ARICI, Aydin1
[1] Department of Obstetrics, Gynecology, and Reproductive Sciences, Yale University School of Medicine, New Haven, Connecticut 06520-8063, United States - Source
The Journal of clinical endocrinology and metabolism. 2006, Vol 91, Num 12, pp 4995-5001, 7 p ; ref : 56 ref
- CODEN
- JCEMAZ
- ISSN
- 0021-972X
- Scientific domain
- Endocrinology; Nutrition, obesity, metabolic disorders
- Publisher
- Endocrine Society, Bethesda, MD
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Apoptose Artère coronaire Cellule endothéliale Endocrinologie Estradiol Facteur nécrose tumorale Homme Ligand Fas Mort cellulaire Cytokine Hormone ovarienne Hormone stéroïde sexuelle Oestrogène
- Keyword (en)
- Apoptosis Coronary artery Endothelial cell Endocrinology Estradiol Tumor necrosis factor Human Fas ligand Cell death Cytokine Ovarian hormone Sex steroid hormone Estrogen
- Keyword (es)
- Apoptosis Arteria coronaria Célula endotelial Endocrinología Estradiol Factor necrosis tumoral Hombre Ligando Fas Muerte celular Citoquina Hormona ovárica Hormona esteroide sexual Estrógeno
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A28 Vertebrates: endocrinology
- Discipline
- Endocrinopathies Vertebrates : endocrinology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 18367761
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1210/jc.2006-1225 
https://dx.doi.org/10.1210/jc.2006-1225
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