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Insulin-like growth factor-I is an important antiapoptotic factor for rat leydig cells during postnatal development

Author
COLON, Eugenia1 ; ZAMAN, Farasat1 ; AXELSON, Magnus2 ; LARSSON, Olle3 ; CARLSSON-SKWIRUT, Christine1 ; SVECHNIKOV, Konstantin V1 ; SÖDER, Olle1
[1] Department of Woman and Child Health, Paediatric Endocrinology Unit, Astrid Lindgren Children's Hospital, Sweden
[2] Department of Clinical Chemistry, Division of Cellular and Molecular Tumor Pathology, Karolinska Institute and University Hospital, 17176 Stockholm, Sweden
[3] Department of Oncology and Pathology, Division of Cellular and Molecular Tumor Pathology, Karolinska Institute and University Hospital, 17176 Stockholm, Sweden
Source

Endocrinology (Philadelphia). 2007, Vol 148, Num 1, pp 128-139, 12 p ; ref : 47 ref

CODEN
ENDOAO
ISSN
0013-7227
Scientific domain
Endocrinology
Publisher
Endocrine Society, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Animal Cellule Leydig Développement postnatal Facteur croissance IGF1 Rat Appareil génital mâle Mammalia Rodentia Testicule Vertebrata
Keyword (en)
Animal Leydig cell Postnatal development Insulin like growth factor 1 Rat Male genital system Mammalia Rodentia Testicle Vertebrata
Keyword (es)
Animal Célula Leydig Desarrollo postnatal Factor crecimiento IGF1 Rata Aparato genital macho Mammalia Rodentia Testículo Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A28 Vertebrates: endocrinology

Discipline
Vertebrates : endocrinology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
18420044

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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