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Regulation of tumour necrosis factor (TNF) induced apoptosis by soluble TNF receptors in Helicobacter pylori infection

Author
SHIBATA, J1 ; GOTO, H1 ; ARISAWA, T1 ; NIWA, Y1 ; HAYAKAWA, T1 ; NAKAYAMA, A1 ; MORI, N2
[1] Second Department of Internal Medicine, Nagoya University School of Medicine, Nagoya, Japan
[2] Department of Pathology, Nagoya University School of Medicine, Nagoya, Japan
Source

Gut. 1999, Vol 45, Num 1, pp 24-31 ; ref : 36 ref

CODEN
GUTTAK
ISSN
0017-5749
Scientific domain
Gastroenterology
Publisher
BMJ, London
Publication country
United Kingdom
Document type
Article
Language
English
Keyword (fr)
Apoptose Bactériose Cytoprotecteur Exploration Facteur nécrose tumorale Facteur soluble Gastrite Helicobacter pylori Homme Mort cellulaire Récepteur Sécrétion Appareil digestif pathologie Bactérie Biologie cellulaire Estomac pathologie Infection Spirillaceae Spirillales
Keyword (en)
Apoptosis Bacteriosis Cytoprotector Exploration Tumor necrosis factor Soluble factor Gastritis Helicobacter pylori Human Cell death Receiver Secretion Digestive diseases Bacteria Cell biology Gastric disease Infection Spirillaceae Spirillales
Keyword (es)
Apoptosis Bacteriosis Citoprotector Exploración Factor necrosis tumoral Factor soluble Gastritis Helicobacter pylori Hombre Muerte celular Receptor Secreción Aparato digestivo patología Bacteria Biología celular Estómago patología Infección Spirillaceae Spirillales
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B05 Infectious diseases / 002B05B Bacterial diseases / 002B05B02 Human bacterial diseases / 002B05B02F Bacterial diseases of the digestive system and abdomen

Discipline
Infectious pathology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1845078

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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