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Inhibition of HtrA2/Omi ameliorates heart dysfunction following ischemia/ reperfusion injury in rat heart in vivo

Author
SHENUARIN BHUIYAN, Md1 ; FUKUNAGA, Kohji1 2
[1] Department of Pharmacology, Graduate School of Pharmaceutical Sciences, Tohoku University, Sendai, Japan
[2] Tohoku University 21st Century COE Program CRESCENDO, Sendai, Japan
Source

European journal of pharmacology. 2007, Vol 557, Num 2-3, pp 168-177, 10 p ; ref : 1 p.1/4

CODEN
EJPHAZ
ISSN
0014-2999
Scientific domain
Pharmacology drugs
Publisher
Elsevier, Amsterdam
Publication country
Netherlands
Document type
Article
Language
English
Author keyword
Cardioprotection HtrA2 (High temperature requirement A2) Ischemia/reperfusion UCF-101 XIAP (X-linked inhibitor of apoptosis protein)
Keyword (fr)
Animal Apoptose Cardioprotecteur Coeur In vivo Inhibiteur protéique Ischémie reperfusion Mort cellulaire Rat Température Trouble fonctionnel Appareil circulatoire Mammalia Rodentia Vertebrata
Keyword (en)
Animal Apoptosis Cardioprotective agent Heart In vivo Protein inhibitor Ischemia reperfusion Cell death Rat Temperature Dysfunction Circulatory system Mammalia Rodentia Vertebrata
Keyword (es)
Animal Apoptosis Cardioprotector Corazón In vivo Inhibidor proteico Isquemia reperfusión Muerte celular Rata Temperatura Trastorno funcional Aparato circulatorio Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B12 Cardiology. Vascular system

Discipline
Cardiology. Circulatory system General pharmacology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
18497281

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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