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Inhibition of PPAR-α activity in mice with cardiac-restricted expression of tumor necrosis factor : potential role of TGF-β/Smad3

Author
SEKIGUCHI, Kenichi1 ; QI TIAN1 ; ISHIYAMA, Masakuni1 ; BURCHFIELD, Jana1 ; FENG GAO1 ; MANN, Douglas L1 ; BARGER, Philip M1
[1] Winters Center for Heart Failure Research, Department of Medicine, Baylor College of Medicine, Houston, Texas, United States
Source

American journal of physiology. Heart and circulatory physiology. 2007, Vol 61, Num 3 ; H1443-H1451 ; ref : 37 ref

CODEN
AJPPDI
ISSN
0363-6135
Scientific domain
Cardiology, blood circulation, phlebology; Physiology, morphology
Publisher
American Physiological Society, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Author keyword
genes inflammation metabolism signal transduction
Keyword (fr)
Appareil circulatoire Facteur croissance transformant β Facteur nécrose tumorale Gène Inflammation Inhibition Métabolisme Récepteur PPAR Souris Transduction signal Tumeur coeur Appareil circulatoire pathologie Cardiopathie Cytokine Mammalia Rodentia Vertebrata
Keyword (en)
Circulatory system Transforming growth factor β Tumor necrosis factor Gene Inflammation Inhibition Metabolism Peroxisome proliferator activated receptor Mouse Signal transduction Heart tumor Cardiovascular disease Heart disease Cytokine Mammalia Rodentia Vertebrata
Keyword (es)
Aparato circulatorio Factor crecimiento transformante β Factor necrosis tumoral Gen Inflamación Inhibición Metabolismo Receptor PPAR Ratón Transducción señal Tumor del corazón Aparato circulatorio patología Cardiopatía Citoquina Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A22 Vertebrates: cardiovascular system

Discipline
Vertebrates : cardiovascular system
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
18626079

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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