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Combined loss of Hey1 and HeyL causes congenital heart defects because of impaired epithelial to mesenchymal transition

Author
FISCHER, Andreas1 ; STEIDL, Christian1 ; WAGNER, Toni U1 ; LANG, Esra2 ; JAKOB, Peter M2 ; FRIEDL, Peter3 4 ; KNOBELOCH, Klaus-Peter5 ; GESSLER, Manfred1 3
[1] Department of Physiological Chemistry I, Biocenter, University of Würzburg, Germany
[2] Experimental Physics 5, University of Würzburg, Germany
[3] Rudolf-Virchow Center, DFG Center for Experimental Biomedicine, University of Würzburg, Germany
[4] Department of Dermatology, University of Würzburg, Germany
[5] Institute of Molecular Pharmacology, Department of Molecular Genetics, Berlin, Germany
Source

Circulation research. 2007, Vol 100, Num 6, pp 856-863, 8 p ; ref : 44 ref

CODEN
CIRUAL
ISSN
0009-7330
Scientific domain
Cardiology, blood circulation, phlebology
Publisher
Lippincott, Hagerstown, MD
Publication country
United States
Document type
Article
Language
English
Author keyword
Hey2 HeyL Heyl Notch epithelial to mesenchymal transition
Keyword (fr)
Appareil circulatoire Cardiopathie congénitale Mammalia Perte Appareil circulatoire pathologie Vertebrata
Keyword (en)
Circulatory system Congenital cardiopathy Mammalia Loss Cardiovascular disease Vertebrata
Keyword (es)
Aparato circulatorio Cardiopathía congénita Mammalia Pérdida Aparato circulatorio patología Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A22 Vertebrates: cardiovascular system

Discipline
Vertebrates : cardiovascular system
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
18653949

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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