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YC-1 inhibits HIF-1 expression in prostate cancer cells: contribution of Akt/NF-κB signaling to HIF-1α accumulation during hypoxia

Author
SUN, H.-L1 ; LIU, Y.-N1 ; HUANG, Y.-T1 ; PAN, S.-L1 ; HUANG, D.-Y1 ; GUH, J.-H2 ; LEE, F.-Y3 ; KUO, S.-C4 ; TENG, C.-M1
[1] Pharmacological Institute, College of Medicine, National Taiwan University, Taipei, Taiwan, Province of China
[2] School of Pharmacy, College of Medicine, National Taiwan University, Taipei, Taiwan, Province of China
[3] Yung-Shin Pharmaceutical Industry Co. Ltd., China Medical College, Taichung, Taiwan, Province of China
[4] Graduate Institute of Pharmaceutical Chemistry, China Medical College, Taichung, Taiwan, Province of China
Source

Oncogene (Basingstoke). 2007, Vol 26, Num 27, pp 3941-3951, 11 p ; ref : 1 p.3/4

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
Akt HIF-1 NF-KB YC-1 prostate cancer
Keyword (fr)
Akt protein kinase Cancer prostate Carcinogenèse Cellule tumorale Expression génique Facteur transcription HIF1 Facteur transcription NFκB Hypoxie Inhibiteur Oxygène Transduction signal Tumeur maligne Appareil génital mâle pathologie Appareil urinaire pathologie Prostate pathologie
Keyword (en)
Akt protein kinase Prostate cancer Carcinogenesis Tumor cell Gene expression Transcription factor HIF1 Transcription factor NFκB Hypoxia Inhibitor Oxygen Signal transduction Malignant tumor Male genital diseases Urinary system disease Prostate disease
Keyword (es)
Akt protein kinase Cáncer de la próstata Carcinogénesis Célula tumoral Expresión genética Factor transcripción HIF1 Factor transcripción NFκB Hipoxia Inhibidor Oxígeno Transducción señal Tumor maligno Aparato genital macho patología Aparato urinario patología Prostata patología
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B14 Nephrology. Urinary tract diseases / 002B14D Tumors of the urinary system / 002B14D02 Urinary tract. Prostate gland

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B20 Gynecology. Andrology. Obstetrics / 002B20B Male genital diseases / 002B20B02 Tumors

Discipline
Gynecology. Andrology. Obstetrics Molecular and cell biology Nephrology. Urinary tract diseases
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
18815401

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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