Estradiol attenuates hyperoxia-induced cell death in the developing white matter
- Author
- GERSTNER, Bettina1 2 ; SIFRINGER, Marco1 3 ; DZIETKO, Mark1 ; SCHÜLLER, Alexandra1 ; LEE, Joan2 ; SIMONS, Sinno1 ; OBLADEN, Michael1 ; VOLPE, Joseph J2 ; ROSENBERG, Paul A2 ; FELDERHOFF-MUESER, Ursula1
[1] Department of Neonatology, Charité Campus Virchow-Klinikum, Berlin, Germany
[2] Department of Neurology and Neurobiology Program, Children's Hospital Boston, Boston, MA, United States
[3] Department of Pediatric Neurology, Children's Hospital, Medical Faculty Carl Gustav Carus, Technical University Dresden, Dresden, Germany - Source
Annals of neurology. 2007, Vol 61, Num 6, pp 562-573, 12 p ; ref : 53 ref
- Publisher
- Willey-Liss, Hoboken
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Keyword (fr)
- Estradiol Hyperoxie provoquée Mort cellulaire Substance blanche Système nerveux pathologie
- Keyword (en)
- Estradiol Induced hyperoxia Cell death White matter Nervous system diseases
- Keyword (es)
- Estradiol Hiperoxia provocada Muerte celular Substancia blanca Sistema nervioso patología
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02B Neuropharmacology / 002B02B10 Neuroprotective agent
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02U Drug toxicity and drugs side effects treatment / 002B02U01 Toxicity: nervous system and muscle
- Discipline
- Drugs toxicity and drug intoxications Neurology Pharmacological treatments Toxicology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 18909321
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1002/ana.21118 
https://dx.doi.org/10.1002/ana.21118
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