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Raloxifene acutely reduces glutamate-induced intracellular calcium increase in cultured rat cortical neurons via inhibition of high-voltage-activated calcium current

Author
HUANG, Y1 2 ; HUANG, Y. L2 ; LAI, B2 ; ZHENG, P2 ; ZHU, Y. C1 ; YAO, T1 2
[1] National Laboratory of Medical Neurobiology, Shanghai Medical Col lege, Fudan University, Shanghai, China
[2] Department of Physiology and Pathophysiology, Fudan University Shanghai Medical College, 138 Yixueyuan Road, Shanghai 200032, China
Source

Neuroscience. 2007, Vol 147, Num 2, pp 334-341, 8 p ; ref : 3/4 p

CODEN
NRSCDN
ISSN
0306-4522
Scientific domain
Neurology; Physiology, morphology
Publisher
Elsevier, Oxford
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
cortical neurons glutamate intracellular calcium selective estrogen receptor modulator voltage-activated calcium channel
Keyword (fr)
Animal Calcium Canal ionique Courant ionique Glutamate In vitro Intracellulaire Neurone Raloxifène Rat Récepteur oestrogène Aminoacide excitateur Mammalia Neurotransmetteur Rodentia Vertebrata
Keyword (en)
Animal Calcium Ionic channel Ionic current Glutamate In vitro Intracellular Neuron Raloxifene Rat Estrogen receptor Excitatory aminoacid Mammalia Neurotransmitter Rodentia Vertebrata
Keyword (es)
Animal Calcio Canal iónico Corriente iónica Glutamato In vitro Intracelular Neurona Raloxifeno Rata Receptor estrógeno Aminoácido excitador Mammalia Neurotransmisor Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A25 Vertebrates: nervous system and sense organs

Discipline
Vertebrates : nervous system and sense organs
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
18919591

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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