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Doxorubicin induces male germ cell apoptosis in rats

Author
SHINODA, K1 ; MITSUMORI, K2 ; YASUHARA, K2 ; UNEYAMA, C2 ; ONODERA, H2 ; HIROSE, M2 ; UEHARA, M3
[1] Pathology Unit, Hita Research Laboratories, Chemicals Inspection and Testing Institute, 3-822 Ishii-machi, Hita-shi, Oita 877-0061, Japan
[2] Division of Pathology, National Institute of Health Sciences, 1-18-1 Kamiyoga, Setagaya-ku, Tokyo 158-8501, Japan
[3] Department of Veterinary Anatomy, Tottori University, 4-101 Minami, Koyama-cho, Tottori 680-8553, Japan
Source

Archives of toxicology. 1999, Vol 73, Num 4-5, pp 274-281 ; ref : 45 ref

CODEN
ARTODN
ISSN
0340-5761
Scientific domain
Toxicology
Publisher
Springer, Berlin
Publication country
Germany
Document type
Article
Language
English
Keyword (fr)
Animal Anthracyclines Anticancéreux Apoptose Doxorubicine Mécanisme action Rat Testicule Toxicité Ultrastructure Voie intraveineuse Appareil génital mâle pathologie Mammalia Rodentia Vertebrata
Keyword (en)
Animal Anthracyclins Antineoplastic agent Apoptosis Doxorubicin Mechanism of action Rat Testicle Toxicity Ultrastructure Intravenous administration Male genital diseases Mammalia Rodentia Vertebrata
Keyword (es)
Animal Antraciclinas Anticanceroso Apoptosis Doxorubicina Mecanismo acción Rata Testículo Toxicidad Ultraestructura Vía intravenosa Aparato genital macho patología Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02U Drug toxicity and drugs side effects treatment / 002B02U06 Toxicity: urogenital system

Discipline
Drugs toxicity and drug intoxications Toxicology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1899793

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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