E2F-1 overexpression in cardiomyocytes induces downregulation of p21CIP1 and p27KIP1 and release of active cyclin-dependent kinases in the presence of insulin-like growth factor I
- Author
- VON HARSDORF, R1 ; HAUCK, L1 ; MEHRHOF, F1 ; WEGENKA, U1 ; CARDOSO, M. C2 ; DIETZ, R1
[1] Department of Cardiology, Franz Volhard Clinic, Humboldt University, Berlin, Germany
[2] Max Delbrück Center for Molecular Medicine, Berlin, Germany - Source
Circulation research. 1999, Vol 85, Num 2, pp 128-136 ; ref : 46 ref
- CODEN
- CIRUAL
- ISSN
- 0009-7330
- Scientific domain
- Cardiology, blood circulation, phlebology
- Publisher
- Lippincott, Hagerstown, MD
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Animal Apoptose Coeur Cycle cellulaire Cycline Facteur croissance IGF1 Facteur transcription Inhibiteur enzyme Kinase Myocyte Rat Surexpression génique Facteur transcription E2F Appareil circulatoire Enzyme Mammalia Mort cellulaire Rodentia Transferases Vertebrata
- Keyword (en)
- Animal Apoptosis Heart Cell cycle Cyclin Insulin like growth factor 1 Transcription factor Enzyme inhibitor Kinase Myocyte Rat Gene overexpression Transcription factor E2F Circulatory system Enzyme Mammalia Cell death Rodentia Transferases Vertebrata
- Keyword (es)
- Animal Apoptosis Corazón Ciclo celular Cyclina Factor crecimiento IGF1 Factor transcripción Inhibidor enzima Kinase Miocito Rata Sobreexpressión genética Aparato circulatorio Enzima Mammalia Muerte celular Rodentia Transferases Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A22 Vertebrates: cardiovascular system / 002A22C Heart
- Discipline
- Vertebrates : cardiovascular system
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 1920145
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1161/01.RES.85.2.128 
https://dx.doi.org/10.1161/01.RES.85.2.128
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