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Aluminum exposure decreases dopamine D1 and D2 receptor expression in mouse brain

Author
KIM, Sunyoun1 ; NAM, Jungmin2 ; KIM, Kisok2
[1] Department of Safety Evaluation, Biotoxtech Company, Chungbuk 363-883, Korea, Republic of
[2] Department of Public Health, Keimyung University, Taegu 704-701, Korea, Republic of
Source

Human & experimental toxicology. 2007, Vol 26, Num 9, pp 741-746, 6 p ; ref : 31 ref

ISSN
0960-3271
Scientific domain
Toxicology
Publisher
Arnold, London
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
dopaminergic neurotransmission neurodegenerative disease neurotoxicity rostrocaudal extent
Keyword (fr)
Aluminium Animal Cerveau Diminution Dopamine Encéphale Exposition Neurotoxicité Neurotransmission Pathologie du système nerveux Récepteur dopaminergique D1 Récepteur dopaminergique D2 Souris Système nerveux Toxicité Catécholamine Mammalia Neurotransmetteur Rodentia Système nerveux central Vertebrata
Keyword (en)
Aluminium Animal Brain Decrease Dopamine Encephalon Exposure Neurotoxicity Neurotransmission Nervous system diseases D1 Dopamine receptor D2 Dopamine receptor Mouse Nervous system Toxicity Catecholamine Mammalia Neurotransmitter Rodentia Central nervous system Vertebrata
Keyword (es)
Aluminio Animal Cerebro Disminución Dopamina Encéfalo Exposición Neurotoxicidad Neurotransmisión Sistema nervioso patología Receptor dopaminérgico D1 Receptor dopaminérgico D2 Ratón Sistema nervioso Toxicidad Catecolamina Mammalia Neurotransmisor Rodentia Sistema nervioso central Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B03 Toxicology / 002B03L Chemical and industrial products toxicology. Toxic occupational diseases / 002B03L05 Metals and various inorganic compounds

Discipline
Toxicology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
19229257

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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