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A role of estrogen/ERα signaling in BRCA1-associated tissue-specific tumor formation

Author
LI, W1 ; XIAO, C1 ; VONDERHAAR, B. K2 ; DENG, C.-X1
[1] Genetics of Development and Disease Branch, 10/9N105, NIDDK, National Institutes of Health, Bethesda, MD, United States
[2] Mammary Biology and Tumorigenesis Laboratory, National Cancer Institute, National Institutes of Health, Bethesda, MD, United States
Source

Oncogene (Basingstoke). 2007, Vol 26, Num 51, pp 7204-7212, 9 p ; ref : 1 p

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
BRCA1 ERK1/2 ERα cyclin D1 mammary tumors
Keyword (fr)
Carcinogenèse Cycline D1 Extracellular signal-regulated protein kinase Glande mammaire Gène BRCA1 Gène suppresseur tumeur Oestrogène Récepteur oestrogène Tissu Tumeur Enzyme Hormone ovarienne Hormone stéroïde sexuelle
Keyword (en)
Carcinogenesis Cyclin D1 Extracellular signal-regulated protein kinase Mammary gland BRCA1 gene Tumor suppressor gene Estrogen Estrogen receptor Tissue Tumor Enzyme Ovarian hormone Sex steroid hormone
Keyword (es)
Carcinogénesis Ciclina D1 Extracellular signal-regulated protein kinase Glándula mamaria Gen BRCA1 Gen supresor tumor Estrógeno Receptor estrógeno Tejido Tumor Enzima Hormona ovárica Hormona esteroide sexual
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
19237336

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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