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The effect of antimicrotubule agents on signal transduction pathways of apoptosis : a review

Author
WANG, L. G1 ; LIU, X. M1 ; KREIS, W1 ; BUDMAN, D. R1
[1] Don Monti Division of Medical Oncology, Department of Medicine, New York University School of Medicine, North Shore University Hospital, Manhasset, New York 11030, United States
Source

Cancer chemotherapy and pharmacology. 1999, Vol 44, Num 5, pp 355-361 ; ref : 89 ref

CODEN
CCPHDZ
ISSN
0344-5704
Scientific domain
Medical oncology; Pharmacology drugs
Publisher
Springer, Berlin
Publication country
Germany
Document type
Article
Language
English
Keyword (fr)
Alcaloïde Anticancéreux Apoptose Article synthèse Colchicine Docétaxel Estramustine Gène onc Gène suppresseur tumeur Microtubule Mécanisme action Paclitaxel Phosphorylation Protein kinase Taxane dérivé Transduction signal Vincristine Vinorelbine Chlorméthine dérivé Gène bcl2 Gène p53 Gène raf Gène ras gène p21 Enzyme Transferases
Keyword (en)
Alkaloid Antineoplastic agent Apoptosis Review Docetaxel Estramustine Onc gene Tumor suppressor gene Microtubule Mechanism of action Paclitaxel Phosphorylation Protein kinase Taxane derivatives Signal transduction Vincristine Vinorelbine Enzyme Transferases
Keyword (es)
Alcaloide Anticanceroso Apoptosis Artículo síntesis Docetaxel Estramustina Gen onc Gen supresor tumor Microtúbulo Mecanismo acción Paclitaxel Fosforilación Protein kinase Taxane derivado Transducción señal Vincristina Vinorelbina Enzima Transferases
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02R Antineoplastic agents / 002B02R01 General aspects

Discipline
Pharmacological treatments
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1954019

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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