Selective reduction and proliferation of teh CD4+ and CD8+ T cell subsets with bispecific monoclonal antibodies : evidence for inter-T cell-mediated cytolysis
- Author
- WONG, J. T; COLVIN, R. B
Massachusetts gen. hosp., dep. medicine immunopathology unit, allergy/immunology unit, Boston MA 02114, United States - Source
Clinical immunology and immunopathology (Print). 1991, Vol 58, Num 2, pp 236-250, 15 p ; ref : 35 ref
- Publisher
- Academic Press, Boston
/
Academic Press, New York, NY
/
Academic Press, San Diego, CA
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Anticorps monoclonal Cytolyse Homme Interaction cellulaire Lymphocyte T Marqueur biologique Multiplication cellulaire Mécanisme Sous population cellulaire Spécificité Sélection Anticorps bispécifique Antigène CD3 Antigène CD4 Antigène CD8
- Keyword (en)
- Monoclonal antibody Cytolysis Human Cell cell interaction T-Lymphocyte Biological marker Cell proliferation Mechanism Cell subpopulation Specificity Selection
- Keyword (es)
- Anticuerpo monoclonal Citólisis Hombre Interacción celular Linfocito T Marcador biológico Multiplicación celular Mecanismo Subpoblación celular Especificidad Selección
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C03 Lymphoid cells: ontogeny, maturation, markers, receptors, circulation and recirculation
- Discipline
- Fundamental immunology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 19712927
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1016/0090-1229(91)90139-2 
https://dx.doi.org/10.1016/0090-1229(91)90139-2
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