The TCL1 oncoprotein inhibits activation-induced cell death by impairing PKCO and ERK pathways
- Author
- DESPOUY, Gilles1 2 ; JOINER, Marjorie1 2 ; LE TORIELLEC, Emilie1 2 ; WEIL, Robert3 ; HENRI STERN, Marc1 2
[1] Institut Curie, Centre de Recherche, Paris, France
[2] Inserm U830, Paris, France
[3] Centre National de la Recherche Scientifique (CNRS) URA2582, Institut Pasteur, Paris, France - Source
Blood. 2007, Vol 110, Num 13, pp 4406-4416, 11 p ; ref : 57 ref
- ISSN
- 0006-4971
- Scientific domain
- Medical oncology; Hematology
- Publisher
- The Americain Society of Hematology, Washington, DC
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Apoptose Extracellular signal-regulated protein kinase Gène onc cellulaire Homme Inhibiteur Leucémie Lignée cellulaire établie Lymphocyte T Lymphome non hodgkinien Mort cellulaire Protein kinase C Protooncogène Transduction signal Gène TCL1 Lignée Jurkat Lignée SUPT11 Voie de signalisation Cancer Enzyme Génétique Hémopathie maligne Syndrome lymphoprolifératif Transferases
- Keyword (en)
- Apoptosis Extracellular signal-regulated protein kinase C-Onc gene Human Inhibitor Leukemia Established cell line T-Lymphocyte Non Hodgkin lymphoma Cell death Protein kinase C Protooncogene Signal transduction Signaling pathway Cancer Enzyme Genetics Malignant hemopathy Lymphoproliferative syndrome Transferases
- Keyword (es)
- Apoptosis Extracellular signal-regulated protein kinase Gen onc celular Hombre Inhibidor Leucemia Línea celular establecida Linfocito T Linfoma no Hodgkin Muerte celular Protein kinase C Protooncogen Transducción señal Cáncer Enzima Genética Hemopatía maligna Linfoproliferativo síndrome Transferases
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B19 Hematologic and hematopoietic diseases / 002B19B Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis
- Discipline
- Blood diseases
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 19920133
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1182/blood-2006-11-059501 
https://dx.doi.org/10.1182/blood-2006-11-059501
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