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ACTIVATION OF GABA RECEPTORS ATTENUATES NEURONAL APOPTOSIS THROUGH INHIBITING THE TYROSINE PHOSPHORYLATION OF NR2A BY Src AFTER CEREBRAL ISCHEMIA AND REPERFUSION

Author
ZHANG, F1 ; LI, C1 ; WANG, R1 ; HAN, D1 ; ZHANG, Q.-G1 ; ZHOU, C1 ; YU, H.-M1 ; ZHANG, G.-Y1
[1] Research Center for Biochemistry and Molecular Biology and Provincial Key Laboratory of Brain Disease Bioinformation, Xuzhou Medical College, 84 West Huai-hai Road, Xuzhou 221002, Jiangsu, China
Source

Neuroscience. 2007, Vol 150, Num 4, pp 938-949, 12 p ; ref : 1 p.1/2

CODEN
NRSCDN
ISSN
0306-4522
Scientific domain
Neurology; Physiology, morphology
Publisher
Elsevier, Oxford
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
GABA receptor NMDA receptor Src baclofen cerebral ischemia muscimol
Keyword (fr)
Apoptose Baclofène Encéphale Ischémie Phosphorylation Récepteur NMDA Récepteur gabaergique Tyrosine Mort cellulaire Pathologie de l'appareil circulatoire Récepteur glutamate Système nerveux central
Keyword (en)
Apoptosis Baclofen Encephalon Ischemia Phosphorylation NMDA receptor Gabaergic receptor Tyrosine Cell death Cardiovascular disease Glutamate receptor Central nervous system
Keyword (es)
Apoptosis Baclofeno Encéfalo Isquemia Fosforilación Receptor NMDA Receptor gabaminérgico Tirosina Muerte celular Aparato circulatorio patología Receptor glutámato Sistema nervioso central
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A25 Vertebrates: nervous system and sense organs

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B17 Neurology / 002B17C Vascular diseases and vascular malformations of the nervous system

Discipline
Neurology Vertebrates : nervous system and sense organs
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
19943068

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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