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ATM-dependent nuclear accumulation of IKK-α plays an important role in the regulation of p73-mediated apoptosis in response to cisplatin

Author
YOSHIDA, K1 2 ; OZAKI, T1 ; FURUYA, K1 ; NAKANISHI, M1 ; KIKUCHI, H1 ; YAMAMOTO, H1 ; ONO, S3 ; KODA, T3 ; OMURA, K2 ; NAKAGAWARA, A1
[1] Division of Biochemistry, Chiba Cancer Center Research Institute, Chiba, Japan
[2] Department of Oral and Maxillofacial Surgery, Tokyo Medical and Dental University, Tokyo, Japan
[3] Research Center for Functional Genomics, Hisamitsu Pharmaceutical Co., Inc, Chiba, Japan
Source

Oncogene (Basingstoke). 2008, Vol 27, Num 8, pp 1183-1188, 6 p ; ref : 1/2 p

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
ATM IKK apoptosis cisplatin p73 ubiquitination
Keyword (fr)
Anticancéreux Apoptose Carcinogenèse Cisplatine Régulation Gène TP73 Ubiquitination
Keyword (en)
Antineoplastic agent Apoptosis Carcinogenesis Cisplatin Regulation(control)
Keyword (es)
Anticanceroso Apoptosis Carcinogénesis Cisplatino Regulación
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20143693

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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