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A pivotal role for Mcl-1 in Bortezomib-induced apoptosis

Author
PODAR, K1 ; GOUILL, S. L1 2 ; ANDERSON, K. C1 ; ZHANG, J1 ; OPFERMAN, J. T3 ; ZORN, E4 ; TAI, Y.-T1 ; HIDESHIMA, T1 ; AMIOT, M2 ; CHAUHAN, D1 ; HAROUSSEAU, J.-L2
[1] Department of Medical Oncology, Jerome Lipper Multiple Myeloma Center, Harvard Medical School, Dana-Farber Cancer Institute, Boston, MA, United States
[2] INSERM U0601, Institut de biologie, 9 quai Moncousu and Service d'Hématologie clinique, Hôtel-Dieu CHU de Nantes, Nantes, France
[3] Department of Biochemistry, St Jude Children's Research Hospital, Memphis, TN, United States
[4] Department of Medicine, Center for Hematologic Oncology, Dana-Farber Cancer Institute, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, United States
Source

Oncogene (Basingstoke). 2008, Vol 27, Num 6, pp 721-731, 11 p ; ref : 1 p.1/4

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
Bortezomib Mcl-1 apoptosis
Keyword (fr)
Anticancéreux Apoptose Bortézomib Carcinogenèse
Keyword (en)
Antineoplastic agent Apoptosis Bortezomib Carcinogenesis
Keyword (es)
Anticanceroso Apoptosis Bortezomib Carcinogénesis
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20167482

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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