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Sphingosine-1-Phosphate Reduces CD4+ T-Cell Activation in Type 1 Diabetes Through Regulation of Hypoxia-Inducible Factor Short Isoform I.1 and CD69

Author
SRINIVASAN, Suseela1 ; BOLICK, David T1 ; LUKASHEV, Dmitriy2 ; LAPPAS, Courtney3 ; SITKOVSKY, Michail2 ; LYNCH, Kevin R3 ; HEDRICK, Catherine C1 3
[1] Robert M. Berne Cardiovascular Research Center, University of Virginia, Charlottesville, Virginia, United States
[2] New England Inflammation and Tissue Protection Institute, Northeastern University, Boston, Massachusetts, United States
[3] Department of Pharmacology, University of Virginia, Charlottesville, Virginia, United States
Source

Diabetes (New York, NY). 2008, Vol 57, Num 2, pp 484-493, 10 p ; ref : 30 ref

CODEN
DIAEAZ
ISSN
0012-1797
Scientific domain
Endocrinology; Nutrition, obesity, metabolic disorders
Publisher
American Diabetes Association, Alexandria, VA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Diabète de type 1 Forme moléculaire Hypoxie Lymphocyte T Endocrinopathie Facteur milieu Immunopathologie Maladie autoimmune Oxygène
Keyword (en)
Type 1 diabetes Molecular form Hypoxia T-Lymphocyte Endocrinopathy Environmental factor Immunopathology Autoimmune disease Oxygen
Keyword (es)
Diabetes de tipo 1 Forma molecular Hipoxia Linfocito T Endocrinopatía Factor medio Inmunopatología Enfermedad autoinmune Oxígeno
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance / 002B21E01A Etiopathogenesis. Screening. Investigations. Target tissue resistance

Discipline
Endocrinopathies
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20181231

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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