Specific combinations of HLA-DR2 and DR3 class II haplotypes contribute graded risk for disease susceptibility and autoantibodies in human SLE
- Author
- GRAHAM, Robert R1 ; ORTMANN, Ward2 ; GAFFNEY, Patrick2 ; GREGERSEN, Peter K4 ; CRISWELL, Lindsey A5 ; HARLEY, John B6 ; BEHRENS, Timothy W2 ; RODINE, Peter2 ; ESPE, Karl2 ; LANGEFELD, Carl3 ; LANGE, Ethan3 ; WILLIAMS, Adrienne3 ; BECK, Stephanie3 ; KYOGOKU, Chieko2 ; MOSER, Kathy2
[1] Department of Molecular Biology, Massachusetts General Hospital, Harvard Medical School, Boston, MA, United States
[2] Department of Rheumatology, Center for Immunology, University of Minnesota Medical School, Minneapolis, MN, United States
[3] Department of Public Health Sciences, Wake Forest University School of Medicine, Winston-Salem, NC, United States
[4] Robert S Boas Center for Genomics and Human Genetics, Feinstein Institute for Medical Research, North Shore Long Island Jewish Health System, Manhasset, NY, United States
[5] Department of Medicine, Division of Rheumatology, Rosalind Russell Medical Research Center for Arthritis, University of California, San Francisco, CA, United States
[6] Department of Medicine, Oklahoma Medical Research Foundation and University of Oklahoma Health Sciences Center, Oklahoma City, OK, United States - Source
European journal of human genetics. 2007, Vol 15, Num 8, pp 823-830, 8 p ; ref : 55 ref
- Publisher
- Nature Publishing, Avenel, NJ
- Publication country
- United States
- Document type
- Article
- Language
- English
- Author keyword
- association study human leukocyte antigen systemic lupus erythematosus
- Keyword (fr)
- Antigène histocompatibilité classe I Antigène histocompatibilité classe II Autoanticorps Autoimmunité Déterminisme génétique Facteur risque Génétique Haplotype Homme Lupus érythémateux disséminé Pathogénie Prédisposition Risque Sensibilité Système HLA Système histocompatibilité majeur Association génétique Immunopathologie Maladie autoimmune Maladie de système Pathologie de la peau Pathologie du tissu conjonctif
- Keyword (en)
- Class I histocompatibility antigen Class II histocompatibility antigen Autoantibody Autoimmunity Genetic determinism Risk factor Genetics Haplotype Human Systemic lupus erythematosus Pathogenesis Predisposition Risk Sensitivity HLA-System Major histocompatibility system Immunopathology Autoimmune disease Systemic disease Skin disease Connective tissue disease
- Keyword (es)
- Antígeno histocompatibilidad clase I Antígeno histocompatibilidad clase II Autoanticuerpo Autoinmunidad Determinismo genético Factor riesgo Genética Haplotipo Hombre Lupus eritematoso sistémico Patogenia Predisposición Riesgo Sensibilidad Sistema HLA Sistema histocompatibilidad mayor Inmunopatología Enfermedad autoinmune Enfermedad sistémica Piel patología Tejido conjuntivo patología
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A07 Genetics of eukaryotes. Biological and molecular evolution
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B07 Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B23 Medical genetics / 002B23A General aspects. Genetic counseling
- Discipline
- Eukaryotes genetics. Biological and molecular evolution Medical genetics Molecular and cell biology Sarcoidosis. Granulomatosis of undetermined origin. Connective tissue diseases. Vasculitides
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 20309016
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1038/sj.ejhg.5201827 
https://dx.doi.org/10.1038/sj.ejhg.5201827
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