Skp2 regulates the antiproliferative function of the tumor suppressor RASSF1A via ubiquitin-mediated degradation at the G1-S transition
- Author
- SONG, M. S1 ; SONG, S. J1 ; KIM, S. J1 ; NAKAYAMA, K2 ; NAKAYAMA, K. I3 ; LIM, D.-S1
[1] Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, Korea, Republic of
[2] Division of Developmental Genetics, Center for Translational and Advanced Animal Research on Human Diseases, Tohoku University Graduate School of Medicine, Sendai, Miyagi, Japan
[3] Department of Molecular and Cellular Biology, Medical Institute of Bioregulation, Kyushu University, Fukuoka, Japan - Source
Oncogene (Basingstoke). 2008, Vol 27, Num 22, pp 3176-3185, 10 p ; ref : 1 p
- ISSN
- 0950-9232
- Scientific domain
- Cell biology, histology; Medical oncology; Genetics
- Publisher
- Nature Publishing, Basingstoke
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Author keyword
- G1-S S transition RASSF1A Skp2 cyclin D-Cdk4 ubiquitination
- Keyword (fr)
- Carcinogenèse Cycline D Gène suppresseur tumeur Kinase cycline dépendante Régulation Ubiquitine Ubiquitination Enzyme Transferases
- Keyword (en)
- Carcinogenesis Cyclin D Tumor suppressor gene Cyclin dependent kinase Regulation(control) Ubiquitin Enzyme Transferases
- Keyword (es)
- Carcinogénesis Ciclina D Gen supresor tumor Cyclin dependent kinase Regulación Ubiquitina Enzima Transferases
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes
- Discipline
- Molecular and cell biology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 20374211
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1038/sj.onc.1210971 
https://dx.doi.org/10.1038/sj.onc.1210971
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