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Storage, Expression and Function of Fas Ligand, the Key Death Factor of Immune Cells

Author
LETTAU, Marcus1 ; PAULSEN, Maren1 ; KABELITZ, Dieter1 ; JANSSEN, Ottmar1
[1] Institute of Immunology, University Hospital Schleswig-Holstein Campus Kiel, Michaelisstr. 5, 24105 Kiel, Germany
Source

Current medicinal chemistry. 2008, Vol 15, Num 17, pp 1684-1696, 13 p ; ref : 197 ref

ISSN
0929-8673
Scientific domain
Biochemistry, molecular biology, biophysics; Pharmacology drugs
Publisher
Bentham Science, Schiphol
Publication country
Netherlands
Document type
Article
Language
English
Author keyword
Fas ligand Nck PCH protein family TNF family activation-induced cell death apoptosis reverse signalling secretory lysosome
Keyword (fr)
Apoptose Cellule NK Cytokine Facteur nécrose tumorale Ligand Fas Lymphocyte T Lysosome Mort cellulaire Mécanisme Stockage Système immunitaire Transduction signal
Keyword (en)
Apoptosis Natural killer cell Cytokine Tumor necrosis factor Fas ligand T-Lymphocyte Lysosome Cell death Mechanism Storage Immune system Signal transduction
Keyword (es)
Apoptosis Célula NK Citoquina Factor necrosis tumoral Ligando Fas Linfocito T Lisosoma Muerte celular Mecanismo Almacenamiento Sistema inmunitario Transducción señal
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death

Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06A General aspects

Discipline
Fundamental immunology Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20479065

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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