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ATF4-dependent transcription is a key mechanism in VEGF up-regulation by oxidized phospholipids : critical role of oxidized sn-2 residues in activation of unfolded protein response

Author
OSKOLKOVA, Olga V1 ; AFONYUSHKIN, Taras1 ; LEITNER, Alexander2 ; VON SCHLIEFFEN, Elena1 ; GARGALOVIC, Peter S3 ; LUSIS, Aldons J3 ; BINDER, Bernd R1 ; BOCHKOV, Valery N1
[1] Department of Vascular Biology and Thrombosis Research, Center for Biomolecular Medicine and Pharmacology, Medical University of Vienna, Vienna, Austria
[2] Department of Analytical Chemistry and Food Chemistry, University of Vienna, Vienna, Austria
[3] Department of Medicine, Division of Cardiology, University of California, Los Angeles, United States
Source

Blood. 2008, Vol 112, Num 2, pp 330-339, 10 p ; ref : 53 ref

ISSN
0006-4971
Scientific domain
Medical oncology; Hematology
Publisher
The Americain Society of Hematology, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Activité biologique Athérosclérose Cellule endothéliale Expression génique Facteur croissance endothélium vasculaire Homme Oxydation Pathogénie Phospholipide Transcription Facteur de transcription ATF4 Cytokine Facteur croissance Génétique Pathologie de l'appareil circulatoire Pathologie des vaisseaux sanguins
Keyword (en)
Biological activity Atherosclerosis Endothelial cell Gene expression Vascular endothelium growth factor Human Oxidation Pathogenesis Phospholipid Transcription Activating transcription factor 4 Cytokine Growth factor Genetics Cardiovascular disease Vascular disease
Keyword (es)
Actividad biológica Ateroesclerosis Célula endotelial Expresión genética Factor crecimiento endotelio vascular Hombre Oxidación Patogenia Fosfolípido Transcripción Citoquina Factor crecimiento Genética Aparato circulatorio patología Vaso sanguíneo patología
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B12 Cardiology. Vascular system / 002B12B Blood and lymphatic vessels / 002B12B01 Atherosclerosis (general aspects, experimental research)

Discipline
Cardiology. Circulatory system
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20499321

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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