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The checkpoint kinases Chkl and Chk2 regulate the functional associations between hBRCA2 and Rad51 in response to DNA damage

Author
BAHASSI, E. M1 ; OVESEN, J. L1 ; RIESENBERG, A. L1 ; BERNSTEIN, W. Z1 ; HASTY, P. E2 ; STAMBROOK, P. J1
[1] Department of Cell and Cancer Biology, University of Cincinnati College of Medicine, Cincinnati, OH, United States
[2] Department of Molecular Medicine, Institute of Biotechnology, The University of Texas Health Science Center at San Antonio, San Antonio, TX, United States
Source

Oncogene (Basingstoke). 2008, Vol 27, Num 28, pp 3977-3985, 9 p ; ref : 1/2 p

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
DNA damage Rad51 homologous recombination phosphorylation
Keyword (fr)
Association Carcinogenèse Cycle cellulaire DNA Gène réparation Kinase Lésion Phosphorylation Point contrôle Recombinaison homologue Régulation Enzyme Transferases
Keyword (en)
Association Carcinogenesis Cell cycle DNA Repair gene Kinase Lesion Phosphorylation Control point Homologous recombination Regulation(control) Enzyme Transferases
Keyword (es)
Asociación Carcinogénesis Ciclo celular DNA Gen reparación Kinase Lesión Fosforilación Punto control Recombinación homólogo Regulación Enzima Transferases
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H02 Cell cycle, cell proliferation

Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20502029

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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