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The apoptotic effect of nanosilver is mediated by a ROS-and JNK-dependent mechanism involving the mitochondrial pathway in NIH3T3 cells

Author
HSIN, Yi-Hong1 2 ; CHEN, Chun-Feng1 ; HUANG, Shing1 ; SHIH, Tung-Sheng3 ; LAI, Ping-Shan4 ; CHUEH, Pinju1
[1] Graduate Institute ofBiomedical Sciences, National Chung Hsing University, Taichung 40227, Taiwan, Province of China
[2] Department of Life Sciences, National Chung Hsing University, Taichung 40227, Taiwan, Province of China
[3] Institute of Occupational Safety and Healthy Council of Labor Affairs, Executive Yuan, Taipei 221, Taiwan, Province of China
[4] Department of Chemistry, National Chung Hsing University, Taichung 40227, Taiwan, Province of China
Source

Toxicology letters. 2008, Vol 179, Num 3, pp 130-139, 10 p ; ref : 1 p

CODEN
TOLED5
ISSN
0378-4274
Scientific domain
Toxicology
Publisher
Elsevier Science, Amsterdam / Elsevier Science, Shannon
Publication country
Ireland
Document type
Article
Language
English
Author keyword
Apoptosis JNK Mitochondria-dependent pathway Nanosilver ROS Reactive oxygen species
Keyword (fr)
Apoptose Espèces réactives de l'oxygène Mitochondrie Mitogen-activated protein kinase Mort cellulaire Mécanisme action Radical libre Stress oxydatif Lignée NIH3T3 c-Jun N-terminal kinase Enzyme Transferases
Keyword (en)
Apoptosis Reactive oxygen species Mitochondria Mitogen-activated protein kinase Cell death Mechanism of action Free radical Oxidative stress Enzyme Transferases
Keyword (es)
Apoptosis Especies reactivas del oxígeno Mitocondria Mitogen-activated protein kinase Muerte celular Mecanismo acción Radical libre Estrés oxidativo Enzima Transferases
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B03 Toxicology

Discipline
Toxicology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20513067

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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