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TRAIL-mediated apoptosis in malignant glioma cells is augmented by celecoxib through proteasomal degradation of survivin

Author
GAISER, T1 ; BECKER, M. R2 ; HABEL, A1 ; REUSS, D. E3 ; EHEMANN, V4 ; RAMI, A5 ; SIEGELIN, M. D1
[1] Department of Neuropathology, University Hospital Heidelberg, Im Neuenheimer Feld 220, 69120 Heidelberg, Germany
[2] Department of Dermatology, University Hospital Heidelberg, Vosstrasse 11, 69115 Heidelberg, Germany
[3] Clinical Cooperation Unit Neuropathology, German Cancer Center (DKFZ), Im Neuenheimer Feld 220, 69120 Heidelberg, Germany
[4] Department of Pathology, University Hospital Heidelberg, Im Neuenheimer Feld 220, 69120 Heidelberg, Germany
[5] Institut für Zelluläre und Molekulare Anatomie (III), Klinikum der Johann Wolfgang von Goethe-Universität, Theodor Stem Kai 7, 60590 Frankfurt/Main, Germany
Source

Neuroscience letters. 2008, Vol 442, Num 2, pp 109-113, 5 p ; ref : 32 ref

CODEN
NELED5
ISSN
0304-3940
Scientific domain
Neurology; Physiology, morphology
Publisher
Elsevier, Shannon
Publication country
Ireland
Document type
Article
Language
English
Author keyword
Glioma,TRAIL/Apo2L,Survivin,Celecoxib,Proteasome
Keyword (fr)
Apoptose Cellule gliale Célécoxib Lignée cellulaire Multicatalytic endopeptidase complex Survivine Enzyme Hydrolases Mort cellulaire Névroglie Peptidases
Keyword (en)
Apoptosis Glial cell Celecoxib Cell line Multicatalytic endopeptidase complex Survivin Enzyme Hydrolases Cell death Neuroglia Peptidases
Keyword (es)
Apoptosis Célula glial Celecoxib Línea celular Multicatalytic endopeptidase complex Survivina Enzima Hydrolases Muerte celular Neuroglia Peptidases
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A25 Vertebrates: nervous system and sense organs

Discipline
Vertebrates : nervous system and sense organs
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20573638

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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