AID and RAG1 do not contribute to lymphomagenesis in Eμ c-myc transgenic mice
- Author
- NEPAL, R. M1 ; ZAHEEN, A1 ; BASIT, W1 ; LI, L1 ; BERGER, S. A1 2 ; MARTIN, A1
[1] Department of Immunology, University of Toronto, Medical Sciences Building, Toronto, Canada
[2] Arthritis and Autoimmunity Research Centre, University Health Network, Toronto, Canada - Source
Oncogene (Basingstoke). 2008, Vol 27, Num 34, pp 4752-4756, 5 p ; ref : 3/4 p
- ISSN
- 0950-9232
- Scientific domain
- Cell biology, histology; Medical oncology; Genetics
- Publisher
- Nature Publishing, Basingstoke
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Author keyword
- B cells RAG activation-induced cytidine deaminase c-myc hypermutation lymphoma
- Keyword (fr)
- Carcinogenèse Commutation isotypique Gène onc cellulaire Lymphocyte B Lymphome Protooncogène Souris Activation-induced cytidine deaminase Cancer Hémopathie maligne Mammalia Rodentia Syndrome lymphoprolifératif Vertebrata
- Keyword (en)
- Carcinogenesis Isotype switch C-Onc gene B-Lymphocyte Lymphoma Protooncogene Mouse Cancer Malignant hemopathy Mammalia Rodentia Lymphoproliferative syndrome Vertebrata
- Keyword (es)
- Carcinogénesis Cambio de isotipo Gen onc celular Linfocito B Linfoma Protooncogen Ratón Cáncer Hemopatía maligna Mammalia Rodentia Linfoproliferativo síndrome Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B19 Hematologic and hematopoietic diseases / 002B19B Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis
- Discipline
- Blood diseases Molecular and cell biology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 20588935
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1038/onc.2008.111 
https://dx.doi.org/10.1038/onc.2008.111
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