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Pressure overload induces cardiac hypertrophy in angiotensin II type 1A receptor knockout mice

Author
HARADA, K1 ; KOMURO, I1 ; YAZAKI, Y1 ; SHIOJIMA, I1 ; HAYASHI, D1 ; KUDOH, S1 ; MIZUNO, T1 ; KIJIMA, K2 ; MATSUBARA, H2 ; SUGAYA, T3 ; MURAKAMI, K4
[1] Department of Medicine III, University of Tokyo School of Medicine, Japan
[2] Second Department of Internal Medicine, Kansai Medical University, Osaka, Japan
[3] Lead Generation Research Laboratories, Tanabe Seiyaku Co, Ltd, Osaka, Japan
[4] Institute of Applied Biochemistry, University of Tsukuba, Ibaraki, Japan
Source

Circulation (New York, N.Y.). 1998, Vol 97, Num 19, pp 1952-1959 ; ref : 55 ref

CODEN
CIRCAZ
ISSN
0009-7322
Scientific domain
Cardiology, blood circulation, phlebology
Publisher
Lippincott Williams & Wilkins, Hagerstown, MD
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Angiotensine II Animal Coeur Hypertrophie Microscopie optique Pathogénie Récepteur biologique Souris Anatomopathologie Appareil circulatoire pathologie Cardiopathie Mammalia Rodentia Vertebrata
Keyword (en)
Angiotensin II Animal Heart Hypertrophy Optical microscopy Pathogenesis Biological receptor Mouse Pathology Cardiovascular disease Heart disease Mammalia Rodentia Vertebrata
Keyword (es)
Angiotensina II Animal Corazón Hipertrofia Microscopía óptica Patogenia Receptor biológico Ratón Anatomía patológica Aparato circulatorio patología Cardiopatía Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B12 Cardiology. Vascular system / 002B12A Heart / 002B12A01 Heart failure, cardiogenic pulmonary edema, cardiac enlargement

Discipline
Cardiology. Circulatory system
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
2242332

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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