2-Deoxy-D-ribose induces cellular damage by increasing oxidative stress and protein glycation in a pancreatic β-cell line
- Author
- KOH, Gwanpyo1 2 ; LEE, Dae-Ho1 ; WOO, Jeong-Taek3
[1] Department of Internal Medicine, Jeju National University School of Medicine, Jeju 690-756, Korea, Republic of
[2] Institute of Medical Science, Jeju National University, Jeju 690-756, Korea, Republic of
[3] Department of Endocrinology and Metabolism, Kyung Hee University School of Medicine, Dongdaemoon-gu, Seoul 130-702, Korea, Republic of - Source
Metabolism, clinical and experimental. 2010, Vol 59, Num 3, pp 325-332, 8 p ; ref : 43 ref
- ISSN
- 0026-0495
- Scientific domain
- Endocrinology; Nutrition, obesity, metabolic disorders
- Publisher
- Elsevier, New York, NY
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Cellule Endocrinologie Glycation Lignée cellulaire Pancréas Protéine Ribose Stress oxydatif
- Keyword (en)
- Cell Endocrinology Glycation Cell line Pancreas Protein Ribose Oxidative stress
- Keyword (es)
- Célula Endocrinología Glicación Línea celular Páncreas Proteína Ribosa Estrés oxidativo
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A16 Vertebrates: anatomy and physiology, studies on body, several organs or systems / 002A16E Feeding. Feeding behavior
- Discipline
- Vertebrates : anatomy and physiology, studies on body, several organs or systems
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 22465181
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1016/j.metabol.2009.07.028 
https://dx.doi.org/10.1016/j.metabol.2009.07.028
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