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14-Deoxyandrographolide desensitizes hepatocytes to tumour necrosis factor- alpha-induced apoptosis through calcium-dependent tumour necrosis factor receptor superfamily member 1A release via the NO/cGMP pathway

Author
ROY, D. N1 ; MANDAL, S1 ; SEN, G1 ; MUKHOPADHYAY, S2 ; BISWAS, T1
[1] Cell Biology and Physiology Division, Indian Institute of Chemical Biology, A Unit of Council of Scientific and Industrial Research, Kolkata, India
[2] Chemistry Division, Indian Institute of Chemical Biology, A Unit of Council of Scientific and Industrial Research, Kolkata, India
Source

British journal of pharmacology. 2010, Vol 160, Num 7, pp 1823-1843, 21 p ; ref : 2 p

CODEN
BJPCBM
ISSN
0007-1188
Scientific domain
Pharmacology drugs
Publisher
Nature Publishing Group, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
14-deoxyandrographolide Ca-ATPase TNF-α TNFR1 TNFRSF1A andrographolide apoptosis cGMP calcium hepatocytes nitric oxide
Keyword (fr)
Adenosinetriphosphatase Apoptose Calcium Cytokine Facteur nécrose tumorale α Facteur nécrose tumorale GMP-3',5' Hépatocyte In vitro Libération Monoxyde d'azote Mort cellulaire Récepteur biologique Andrographolide Appareil digestif Elément minéral Enzyme Foie Hydrolases
Keyword (en)
Adenosinetriphosphatase Apoptosis Calcium Cytokine Tumor necrosis factor α Tumor necrosis factor 3',5'-GMP Hepatocyte In vitro Release Nitric oxide Cell death Biological receptor Digestive system Inorganic element Enzyme Liver Hydrolases
Keyword (es)
Adenosinetriphosphatase Apoptosis Calcio Citoquina Factor necrosis tumoral α Factor necrosis tumoral GMP-3',5' Hepatocito In vitro Liberación Nitrógeno monóxido Muerte celular Receptor biológico Aparato digestivo Elemento inorgánico Enzima Hígado Hydrolases
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments

Discipline
General pharmacology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
22989526

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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