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Tumor necrosis factor-α suppresses angiotensinogen expression through formation of a p50/p50 homodimer in human renal proximal tubular cells

Author
SATOU, Ryousuke1 ; MIYATA, Kayoko1 ; KATSURADA, Akemi1 ; NAVAR, L. Gabriel1 ; KOBORI, Hiroyuki1
[1] Department of Physiology, and Hypertension and Renal Center of Excellence, Tulane University Health Sciences Center, New Orleans, Louisiana, United States
Source

American journal of physiology. Cell physiology. 2010, Vol 68, Num 4 ; C750-C759 ; ref : 39 ref

CODEN
AJPCDD
ISSN
0363-6143
Scientific domain
Cell biology, histology; Physiology, morphology
Publisher
American Physiological Society, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Author keyword
human kidney-2 nuclear factor-κB
Keyword (fr)
Facteur nécrose tumorale α Homme Rein Appareil urinaire Cytokine
Keyword (en)
Tumor necrosis factor α Human Kidney Urinary system Cytokine
Keyword (es)
Factor necrosis tumoral α Hombre Riñón Aparato urinario Citoquina
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A19 Vertebrates: skin, associated glands, phaneres, light organs, various exocrine glands (salt gland, uropygial gland...), adipose tissue, connective tissue

Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A23 Vertebrates: osteoarticular system, musculoskeletal system / 002A23A Skeleton and joints

Discipline
Vertebrates : osteoarticular system, musculoskeletal system Vertebrates : skin, associated glands, phaneres, light organs, various exocrine glands, adipose tissue, connective tissue
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
23233656

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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