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Effect of Multikinase Inhibitors on Caspase-Independent Cell Death and DNA Damage in HER2-Overexpressing Breast Cancer Cells

Author
SEOANE, Samuel1 ; MONTERO, Juan Carlos1 ; OCANA, Alberto2 ; PANDIELLA, Atanasio1
[1] Instituto de Biología Molecular y Celular del Cáncer, CSIC-Universidad de Salamanca, Salamanca, Spain
[2] Servicio de Oncología Médica, Complejo Hospitalario Universitario de Albacete, AECC Unit, Albacete, Spain
Source

Journal of the National Cancer Institute. 2010, Vol 102, Num 18, pp 1432-1446, 15 p ; ref : 45 ref

ISSN
0027-8874
Scientific domain
Medical oncology
Publisher
Oxford University Press, Cary, NC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Apoptose Cancer du sein Cancérologie Caspase Cellule tumorale Cysteine endopeptidases Gène erbB2 Gène onc cellulaire Inhibiteur Lésion Mort cellulaire Protooncogène Surexpression génique Récepteur HER2 Cancer Enzyme Hydrolases Pathologie de la glande mammaire Pathologie du sein Peptidases Tumeur maligne
Keyword (en)
Apoptosis Breast cancer Cancerology Caspase Tumor cell Cysteine endopeptidases erbB2 Gene C-Onc gene Inhibitor Lesion Cell death Protooncogene Gene overexpression Human Epidermal growth factor Receptor 2 Cancer Enzyme Hydrolases Mammary gland diseases Breast disease Peptidases Malignant tumor
Keyword (es)
Apoptosis Cáncer del pecho Cancerología Caspase Célula tumoral Cysteine endopeptidases Gen erbB2 Gen onc celular Inhibidor Lesión Muerte celular Protooncogen Sobreexpresión genética Cáncer Enzima Hydrolases Glándula mamaria patología Seno patología Peptidases Tumor maligno
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B04 Tumors

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B20 Gynecology. Andrology. Obstetrics / 002B20E Mammary gland diseases / 002B20E02 Tumors

Discipline
Gynecology. Andrology. Obstetrics Tumours
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
23284302

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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