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Effect of Ha-rasval12 on nm23 Expression, Tumor Formation and Metastasis of the Transformants, and Immunomodulation in Tumor-bearing Mice

Author
HSIEH, Chang-Chi1 2 ; WU, Shan-Ying3 ; LAN, Sheng-Huei3 ; WENG, Tzu-Yu3 ; TSAI, Y. U4 ; LIU, Hsiao-Sheng4 5
[1] Department of Animal Science and Biotechnology, Tunghai University, Taichung, Taiwan, Province of China
[2] School of Chinese Medicine Resources, China Medical University, Taichung, Taiwan, Province of China
[3] Institute of Basic Medical Sciences, College of Medicine, National Cheng Kung University, Tainan, Taiwan, Province of China
[4] Department of Microbiology and Immunology, College of Medicine, National Cheng Kung University, Tainan, Taiwan, Province of China
[5] Center for Gene Regulation and Signal Transduction Research, National Cheng Kung University, Tainan, Taiwan, Province of China
Source

Anticancer research. 2010, Vol 30, Num 9, pp 3585-3592, 8 p ; ref : 24 ref

ISSN
0250-7005
Scientific domain
Medical oncology
Publisher
International Institute of Anticancer Research, Attiki
Publication country
Greece
Document type
Article
Language
English
Author keyword
Ha-ras immunomodulation metastasis nm23
Keyword (fr)
Animal Cancérologie Carcinogenèse Gène onc cellulaire Gène ras Gène suppresseur métastase Immunomodulation Métastase Protooncogène Souris Tumeur maligne Tumorigénicité Cancer Mammalia Rodentia Vertebrata
Keyword (en)
Animal Cancerology Carcinogenesis C-Onc gene Ras gene Metastasis suppressor gene Immunomodulation Metastasis Protooncogene Mouse Malignant tumor Tumorigenicity Cancer Mammalia Rodentia Vertebrata
Keyword (es)
Animal Cancerología Carcinogénesis Gen onc celular Gen ras Gen supresor metástasis Inmunomodulación Metástasis Protooncogen Ratón Tumor maligno Tumorigenicidad Cáncer Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B04 Tumors

Discipline
Tumours
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
23293050

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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