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Silver nanoparticles induce oxidative cell damage in human liver cells through inhibition of reduced glutathione and induction of mitochondria-involved apoptosis

Author
MEI JING PIAO1 ; KYOUNG AH KANG1 ; IN KYUNG LEE2 ; HYE SUN KIM2 ; KIM, Suhkmann3 ; JEONG YUN CHOI4 ; CHOI, Jinhee5 ; JIN WON HYUN1
[1] School of Medicine and Applied Radiological Science Research Institute, Jeju National University, Jeju 690-756, Korea, Republic of
[2] School of Medicine, Seoul National University, Seoul 110-799, Korea, Republic of
[3] Department of Chemistry and Chemistry Institute for Functional Materials, Pusan National University, Pusan 609-735, Korea, Republic of
[4] Department of Pharmacology, School of Medicine, Ewha Womans University, Seoul 158-710, Korea, Republic of
[5] Faculty of Environmental Engineering, University of Seoul, Seoul 130-743, Korea, Republic of
Source

Toxicology letters. 2011, Vol 201, Num 1, pp 92-100, 9 p ; ref : 1 p.1/2

CODEN
TOLED5
ISSN
0378-4274
Scientific domain
Toxicology
Publisher
Elsevier, Shannon
Publication country
Ireland
Document type
Article
Language
English
Author keyword
Apoptosis Glutathione Oxidative stress Silver nanoparticles
Keyword (fr)
Apoptose Argent Foie Glutathion Homme In vitro Induction Inhibiteur Inhibition Mitochondrie Mort cellulaire Nanoparticule Particule ultrafine Stress oxydatif Nanomatériau Appareil digestif Métal transition
Keyword (en)
Apoptosis Silver Liver Glutathione Human In vitro Induction Inhibitor Inhibition Mitochondria Cell death Nanoparticle Ultrafine particle Oxidative stress Digestive system Transition metal
Keyword (es)
Apoptosis Plata Hígado Glutatión Hombre In vitro Inducción Inhibidor Inhibición Mitocondria Muerte celular Nanopartícula Partícula ultrafina Estrés oxidativo Aparato digestivo Metal transición
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B03 Toxicology / 002B03L Chemical and industrial products toxicology. Toxic occupational diseases / 002B03L05 Metals and various inorganic compounds

Discipline
Toxicology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
23861508

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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