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NF-κB antiapoptosis: Induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to suppress caspase-8 activation

Author
WANG, C.-Y1 ; MAYO, M. W2 ; KORNELUK, R. G3 ; GOEDDEL, D. V4 ; BALDWIN, A. S5
[1] Department of Endodontics, School of Dentistry, Lineberger Comprehensive Cancer Center, and Curriculum in Genetics and Molecular Biology, University of North Carolina, Chapel Hill, NC 275997295, United States
[2] Lineberger Comprehensive Cancer Center, University of North Carolina, Chapel Hill, NC 27599-7295, United States
[3] Molecular Genetics Research Laboratory, Children's Hospital of Eastern Ontario, Ottawa, Ontario K1H 8M5, Canada
[4] Tularik, 2 Corporate Drive, South San Francisco, CA 94080, United States
[5] Lineberger Comprehensive Cancer Center, Curriculum in Genetics and Molecular Biology, and Department of Biology. University of North Carolina, Chapel Hill, NC 27599-7295, United States
Source

Science (Washington, D.C.). 1998, Vol 281, Num 5383, pp 1680-1683

CODEN
SCIEAS
ISSN
0036-8075
Scientific domain
Multidisciplinary
Publisher
American Association for the Advancement of Science, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Activité enzymatique Apoptose Expression génique Facteur nécrose tumorale α Gène Mammalia Régulation Transduction signal Caspase Facteur transcription NFκB Cytokine Vertebrata
Keyword (en)
Enzymatic activity Apoptosis Gene expression Tumor necrosis factor α Gene Mammalia Regulation(control) Signal transduction Transcription factor NFκB Cytokine Vertebrata
Keyword (es)
Actividad enzimática Apoptosis Expresión genética Factor necrosis tumoral α Gen Mammalia Regulación Transducción señal Citoquina Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
2391821

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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