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Myostatin promotes the wasting of human myoblast cultures through promoting ubiquitin-proteasome pathway-mediated loss of sarcomeric proteins

Author
LOKIREDDY, Sudarsanareddy1 ; MOULY, Vincent2 ; BUTLER-BROWNE, Gillian2 ; GLUCKMAN, Peter D3 ; SHARMA, Mridula4 ; KAMBADUR, Ravi1 3 ; MCFARLANE, Craig3
[1] School of Biological Sciences, Nanyang Technological University, Singapore
[2] UPMC Université Paris 6, Institut de Myologie, Paris, France
[3] Growth, Development and Metabolism Program, Singapore Institute for Clinical Sciences, Singapore
[4] Yong Loo Lin School of Medicine, Department of Biochemistry, National University of Singapore, Singapore
Source

American journal of physiology. Cell physiology. 2011, Vol 70, Num 6 ; C1316-C1324 ; ref : 40 ref

CODEN
AJPCDD
ISSN
0363-6143
Scientific domain
Cell biology, histology; Physiology, morphology
Publisher
American Physiological Society, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Author keyword
AKT Forkhead Box-O SMAD3 atrogin-1 myostatin-induced human muscle wasting
Keyword (fr)
Akt protein kinase Homme Myoblaste Protéine Ubiquitine
Keyword (en)
Akt protein kinase Human Myoblast Protein Ubiquitin
Keyword (es)
Akt protein kinase Hombre Mioblasto Proteína Ubiquitina
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04J Inflammation

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
24818002

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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