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Modulation of gap junctional intercellular communication by EGF in human kidney epithelial cells

Author
RIVEDAL, E1 ; MOLLERUP, S2 ; HAUGEN, A2 ; VIKHAMAR, G1
[1] Laboratory for Environmental and Occupational Cancer, Institute for Cancer Research, The Norwegian Radium Hospital, 0310 Oslo, Norway
[2] Department of Toxicology, National Institute of Occupational Health, PO Box 8149 Dep, 0033 Oslo, Norway
Source

Carcinogenesis (New York. Print). 1996, Vol 17, Num 11, pp 2321-2328 ; ref : 72 ref

CODEN
CRNGDP
ISSN
0143-3334
Scientific domain
Medical oncology; Genetics; Pharmacology drugs; Toxicology
Publisher
Oxford University Press, Oxford
Publication country
United Kingdom
Document type
Article
Language
English
Keyword (fr)
Appareil urinaire Carcinogenèse Cellule tumorale Cellule épithéliale Connexine Facteur croissance épiderme Homme Jonction communicante Mécanisme action Phase multiple Rein Sous population cellulaire Translation Transport intercellulaire Connexine 43
Keyword (en)
Urinary system Carcinogenesis Tumor cell Epithelial cell Connexin Epidermal growth factor Human Gap junction Mechanism of action Multiple phase Kidney Cell subpopulation Translation Intercellular transport
Keyword (es)
Aparato urinario Carcinogénesis Célula tumoral Célula epitelial Connexina Factor crecimiento epidermi Hombre Unión comunicante Mecanismo acción Fase múltiple Riñón Subpoblación celular Translación Transporte intercelular
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H06 Membrane and intracellular transports

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
2500514

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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